Heredity B/Designer Genes C

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moonwax
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Re: Heredity B/Designer Genes C

Post by moonwax »

1. In which phase of meiosis do cells first become haploid?
2. What is the difference between pyrimidines and purines?
3. What is overdominance?
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EwwPhysics
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Re: Heredity B/Designer Genes C

Post by EwwPhysics »

moonwax wrote: March 20th, 2020, 11:08 pm 1. In which phase of meiosis do cells first become haploid?
2. What is the difference between pyrimidines and purines?
3. What is overdominance?
1.
I guess telophase II? They don't become separate haploid cells until cytokinesis has occurred, I don't know if they would be considered haploid during anaphase II, when the chromosomes have split but they're still in the same cell technically...
2.
Purines are bigger because they have 4 nitrogen atoms while pyrimidines only have 2.
3.
Overdominance is when heterozygous individuals have a higher fitness than homozygous individuals.
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Re: Heredity B/Designer Genes C

Post by moonwax »

EwwPhysics wrote: March 21st, 2020, 7:44 am
moonwax wrote: March 20th, 2020, 11:08 pm 1. In which phase of meiosis do cells first become haploid?
2. What is the difference between pyrimidines and purines?
3. What is overdominance?
1.
I guess telophase II? They don't become separate haploid cells until cytokinesis has occurred, I don't know if they would be considered haploid during anaphase II, when the chromosomes have split but they're still in the same cell technically...
2.
Purines are bigger because they have 4 nitrogen atoms while pyrimidines only have 2.
3.
Overdominance is when heterozygous individuals have a higher fitness than homozygous individuals.
1. Gahhh, okay, next time I really should think more before giving questions... I believe the answer is cytokinesis in meiosis I? I don’t know, but my reasoning is that after meiosis I each cell has only one form of genetic information, just duplicated. I can’t find any definition of haploid on the web besides “half the usual number of chromosomes” or “single set of chromosomes,” though, so this question is kind of tricky... sorry. Here’s the link to a Khan Academy article on meiosis, which I was reading before posting this, but I don’t know... https://www.khanacademy.org/science/bio ... of-meiosis The rest are definitely correct, though! (And I’m probably just wrong on the first question...)
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Re: Heredity B/Designer Genes C

Post by EwwPhysics »

hmmm it just depends on the definition of haploid.


1. What is the Rh factor?
2. How can Rh incompatibility affect pregnancy?

3. What are the loops of DNA in prokaryotes called?

4. What are the roles of untranslated regions of mRNA?
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Re: Heredity B/Designer Genes C

Post by MoMoney$$$;)0) »

EwwPhysics wrote: March 22nd, 2020, 6:14 am hmmm it just depends on the definition of haploid.


1. What is the Rh factor?
2. How can Rh incompatibility affect pregnancy?

3. What are the loops of DNA in prokaryotes called?

4. What are the roles of untranslated regions of mRNA?
It's essentially pH, if it's too high it's bad for you, ChRoMoSoMeS (jKjK), to bE uSeD aS TrNa
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Re: Heredity B/Designer Genes C

Post by EwwPhysics »

MoMoney$$$;)0) wrote: March 22nd, 2020, 3:36 pm
EwwPhysics wrote: March 22nd, 2020, 6:14 am hmmm it just depends on the definition of haploid.


1. What is the Rh factor?
2. How can Rh incompatibility affect pregnancy?

3. What are the loops of DNA in prokaryotes called?

4. What are the roles of untranslated regions of mRNA?
It's essentially pH, if it's too high it's bad for you, ChRoMoSoMeS (jKjK), to bE uSeD aS TrNa
aLL coRrEcT
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MoMoney$$$;)0) (March 23rd, 2020, 9:36 am)
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Re: Heredity B/Designer Genes C

Post by EwwPhysics »

I'd like to apologize for MoMoney$$$;)0) and I's joking around, that's probably what led to inactivity on this thread.
Remember that those questions still have to be answered!
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Re: Heredity B/Designer Genes C

Post by Phenakism »

Oh lmao ok
1.Rh factor is the different blood type system where the positive genotype adds a new rh antigen to your red blood cells. \
2. If the baby and the mother have different rh factors, the mom negative and the baby positive, the mom's antibodies end up attacking the baby and cause anemia. 3. Plasmids? 4. Like the 5'end 3' poly-A tail? The 5' cap helps with ribosome interconnecting and both protect from enzymic degradation
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Re: Heredity B/Designer Genes C

Post by EwwPhysics »

Phenakism wrote: April 20th, 2020, 1:27 pm Oh lmao ok
1.Rh factor is the different blood type system where the positive genotype adds a new rh antigen to your red blood cells. \
2. If the baby and the mother have different rh factors, the mom negative and the baby positive, the mom's antibodies end up attacking the baby and cause anemia. 3. Plasmids? 4. Like the 5'end 3' poly-A tail? The 5' cap helps with ribosome interconnecting and both protect from enzymic degradation
All correct, but for 4 I said They help with mRNA stability (i.e. protection from ribonucleases which degrade), localization, and translational efficiency, you were basically right, I was talking about the regions before the start codon and after the stop codon, and I guess the 5' cap and poly-A tail would be included in that
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Re: Heredity B/Designer Genes C

Post by reninkidney »

1. What are the levels of DNA compaction?

2. How does prokaryotic DNA differ from eukaryotic DNA?

3. What is the spindle checkpoint?
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